Molecular Docking Approach to Identify Potential AntiCandidal Potential of Curcumin.

dc.contributor.authorNidhi Rani
dc.contributor.authorPrerna Sharma
dc.contributor.authorPraveen Kuma
dc.date.accessioned2025-12-18T09:58:24Z
dc.date.available2025-12-18T09:58:24Z
dc.date.issued2020-11-17
dc.description.abstractBackground: Candida albicans is a kind of fungus that can lead to mortality. In the presence of the enzyme Lanosterol-demethylase, Ergosterol, the major sterol in the fungal cell membrane, is the resulting product of Lanosterol (Cytochrome P450DM). Purpose: Azole antifungal drugs target this enzyme as a target enzyme. The work included selecting and modelling the target enzyme. Cucumin’s inhibitory effect on Cytochrome P450 was tested utilising molecular docking experiments. Methods: Chem sketch was used to create compound structures, and Molergo Virtual Docker was used to do molecular docking. Results: All of the curcumin and conventional medicines, such as Ketoconazole, Clotrimazole, and Miconazole, have interaction with 14-demethylase amino acid residues, Haem and water molecules in the target site, as per the docking research.
dc.identifier.issn2321-2217
dc.identifier.issn2321-2225
dc.identifier.otherhttps://doi.org/10.15415/jptrm.2020.82008
dc.identifier.urihttps://demodspace.chitkara.edu.in/handle/123456789/246
dc.language.isoen
dc.publisherChitkara University Publications
dc.subjectAntifungal medications
dc.subjectCandida albicans
dc.subject14α-demethylase
dc.subjectMolecular docking
dc.subjectMolecular modeling
dc.titleMolecular Docking Approach to Identify Potential AntiCandidal Potential of Curcumin.
dc.typeArticle

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